Showing posts with label diet. Show all posts
Showing posts with label diet. Show all posts

Friday, 16 September 2016

The Brain in Super Agers

My Twitter post on a recently published study of brain structure in a group of high performing older adults received quite a bit of attention (see below).

Felicia Sun and colleagues at Massachusetts General Hospital and Harvard Medical School selected an interesting research design.

A group of elderly subjects between the ages of 60 and 80 years were identified as showing "superaging". This was defined as scoring like young adults on two neuropsychological tests: The Long Delay Free Recall measure of the California Verbal Learning Test and part B of the Trail Making Test.

They then imaged the brain structure and function of the super agers using MRI. The results were compared to two groups: elderly adults without superior cognitive performance and younger adults with age-typical neuropsychological performance.

A key finding from their study was that super agers had brain hippocampal volumes greater than typical older adults and this measure was comparable to young adults.

Super agers also had greater brain volumes than typical older adults in the following regions:
  • Anterior temporal cortex
  • Medial frontal cortex
  • Anterior midcingulate cortex

The authors noted in the discussion section:
"We found support for our hypothesis regarding the structural integrity of the default mode and salience networks, with superagers showing much less atrophy than typical older adults in key nodes of these networks, which we refer to as the "superageing signature".
The authors also note their longitudinal study was not able to identify the factors that may play a key role in super agers. Candidate factors include genetic factors, exercise, diet and social activity levels. These factors are likely to be studied in future research.

Readers with more interest in this study can access the free full-text manuscript by clicking the citation link below.

Figure of hippocampus is an iPad screen shot from the app 3D Brain.

Follow me on Twitter @WRY999

Sun FW, Stepanovic MR, Andreano J, Barrett LF, Touroutoglou A, & Dickerson BC (2016). Youthful Brains in Older Adults: Preserved Neuroanatomy in the Default Mode and Salience Networks Contributes to Youthful Memory in Superaging. The Journal of neuroscience : the official journal of the Society for Neuroscience, 36 (37), 9659-9668 PMID: 27629716

Friday, 24 June 2016

Aging and Cognition: Free eBook Resource

Yesterday I posted on Twitter a link to a post on the Dana Foundation website.

In this post, they reviewed highlights from a seminar on cognitive aging sponsored by the American Association for the Advancement of Science (AAAS).

There is a YouTube video of the seminar that I will be reviewing in the next few days and will likely write up a summary of my notes from the video.

One important item I picked up was the recommendation of a free PDF book titled:
Cognitive Aging: Progress in Understanding and Opportunities for Action.

You can access this free aging and cognition book resource by clicking HERE.

The link to the Dana Foundation post is HERE.

Follow me on Twitter (@WRY999) HERE.

Monday, 16 May 2016

Older Fathers: Epigenetic Factors

 A recent review article published in American Journal of Stem Cells summarized current knowledge of epigenetic factors in fathers.

Epigenetic factors are defined as environmental factors that change the expression of genetics.

We know that as men age, changes in genetic structures have effects on their offspring.

Men who have children after age 40 have higher rates of offspring with:

  • schizophrenia
  • autism and autism spectrum disorder
  • birth defects including heart defects, Down syndrome and other chromosomal anomalies
These effects are not completely understood but do appear to be related to age-dependent methylation of DNA.

Older age is not the only epigenetic influence in fathers. Cigarette smoking, heavy alcohol intake, paternal diet and paternal stress all have some evidence of adversely affecting paternal epigenetic effects.

The effect of advanced paternal age on increased risk in offspring is important as the age at first child is increasing for both men and women. Part of older father effect appears related to the association of older parenthood with increased level of education in the population. (see original chart produced above from U.S. Census Bureau data)

You can read more about this review at MedicalXpress HERE.

Follow the author on Twitter @WRY999 HERE.




Monday, 19 January 2015

Mediterranean Diet and Aging

There is a growing research body of evidence to support beneficial effects of a Mediterranean diet on brain health.

In previous posts I have reviewed research on the Mediterranean diet and:

Cognitive Decline

Alzheimer's Disease Prevention

A recent study adds an important element in potential mechanisms for the beneficial effects of the Mediterranean diet.

Marta Crous-Bou and colleauges from Harvard University and the University of Washington published a study of the Mediterranean diet and chromosome telomere length.

This analysis used the large Nurses' Health Study cohort, blood samples and dietary questionnaires. Blood samples were analyzed for chromosome telomere length.

Chromosome telomere length has emerged as a biomarker of aging. DNA chromosome telomere lengths decline with age and shortened telomere lengths are associated with shortened life expectancy. 

The natural process of telomere shortening appears accelerated by inflammation and may be modified by lifestyle behaviors including dietary composition.

In the current study, subjects regularly completed dietary questionnaires. These questionnaires were rated on compliance with the Mediterranean diet on nine variables:

  1. Vegetables
  2. Fruits
  3. Nuts
  4. Whole grains
  5. Legumes
  6. Fish
  7. Monounsaturated fat to saturated fat ratio
  8. Red meat consumption
  9. Alcohol intake

Median values for variables 1-8 were identified and subjects received a score of 1 if they were above the median (below median for red meat consumption). One point was awarded for consuming between 5 and 15 grams of alcohol per day.

Telomere length shortening was associated with older age and heavier cigarette consumption history.

Subjects were grouped into quintiles (20%) groups from lowest to highest Mediterranean diet score.

After adjusting for confounding variables, telomere length was statistically linked to Mediterranean diet score. Those with higher ratings on Mediterranean diet had longer telomeres indicating better aging.

The highest quintile group reported the following mean daily servings for each category: Vegetables (4.3 servings per day), fruits (3.2), whole grains (2.1), fish (0.5), red meat (0.5), legumes (0.6) and nuts (0.5).

The authors note they were unable to identify any single variable that had a strong independent effect. The effect appeared to be a combined global effect of the Mediterranean diet score.

This is an important study because it adds evidence for a biological mechanism for some of the previously reported clinical advantages associated with a Mediterranean diet. 

However, this study is limited by being primarily cross-sectional in design. It does not inform on whether changing diet to a Mediterranean type reduces rate of telomere shortening. 

Readers with more interest in this research can access the free full-text manuscript by clicking on the citation link below.

Photo of foods highlighted in the Mediterranean diet is an original photo from my files.

Follow me on Twitter WRY999

Crous-Bou M, Fung TT, Prescott J, Julin B, Du M, Sun Q, Rexrode KM, Hu FB, & De Vivo I (2014). Mediterranean diet and telomere length in Nurses' Health Study: population based cohort study. BMJ (Clinical research ed.), 349 PMID: 25467028

Wednesday, 20 July 2011

Nuts, Booze, Soy and Cardiovascular Mortality

I have previously published several posts related to diet, cardiovascular mortality and cognitive function.  A recent post looked at the relationship of fiber intake and mortality.  Another post examined adherence to a Mediterranean style diet with reduced risk of cognitive decline.  Now there is another important study published examining diet and risk of cardiovascular mortality and all-cause mortality. 

Cardiovascular disease and mortality are important issues for many of the clinical neuroscience diseases.  Heart disease is often the first issue that comes to mind when thinking about cardiovascular disease.  However, here is just a partial list of clinical neuroscience disorders linked to vascular function and health:
  • Stroke
  • Vascular and Alzheimer's dementia
  • Transient ischemic attacks
  • Vascular neuropathy
A study Akbaraly et al published in The American Journal of Clinical Nutriton examined dietary history and mortality in those participating in the Whitehall II epidemiologic study.  The Whitehall II study is a study of Londoners who worked in one of 20 civil service occupations.  The baseline data was first collected in 1985 through 1988 (when subjects were 35 to 55 years old) but nutritional information was not collected until 1991-1993.  At that time subjects were scored on a composite index of healthy eating called the Alternative Healthy Eating Index (AHEI) composed of the following subscale categories:
  • Vegetables
  • Fruits
  • Nuts and soy
  • Ratio of white meat to red meat
  • Total fiber
  • Trans fat
  • Ratio of unsaturated fats to saturated fats
  • Duration of multivitamin use
  • Alcohol intake
A composite score is calculated from this index with higher scores indicating presumed healthier diet.  The study subjects were grouped in three groups--low AHEI , intermediate AHEI and high AHEI each group representing a third of the sample.  


After 18 years of follow up, approximately 10% of the low AHEI subjects had died compared to 7% of the high AHEI subjects.  Cardiovascular mortality was 42% less for those in the high AHEI group.  All estimates were made after controlling for a variety of potential confounding sociodemographic variables, i.e. gender, BMI, smoking status).


There were two findings that caught my attention from this study.  One was some of the inflammatory and clinical differences in the groups.  The high AHEI group:
  • Had lower serum levels of inflammatory markers C-reactive protein (CRP) and interleukin-6 (IL-6)
  • Had lower levels of dyslipidemias (elevated cholesterol or triglycerides)
  • Had no lower risk of having hypertension or diabetes
A second finding in the study was the examination of the contribution of individual categories to correlation with lower cardiovascular mortality risk.  Only two of the categories correlated with reduced cardiovascular death rates: nuts and soy AHEI scale score and alcohol intake scale score.  Total fiber intake had the next largest individual contribution but failed to be statistically correlate with reduced mortality.


Maximum scores in the nuts and soy category were reached with only one serving per day.  Maximum scores in the alcohol intake category were reached with two drinks per day for men and one drink per day for women. 


This study adds impetus to further study of what are the most important contributing factors of a "healthy diet" when health is measured as reduce risk of mortality from cardiovascular disease and all other causes.


Photo of Carlos Zambrano pitching for the Chicago Cubs courtesy of Yates Photography.
  
Akbaraly TN, Ferrie JE, Berr C, Brunner EJ, Head J, Marmot MG, Singh-Manoux A, Ritchie K, Shipley MJ, & Kivimaki M (2011). Alternative Healthy Eating Index and mortality over 18 y of follow-up: results from the Whitehall II cohort. The American journal of clinical nutrition, 94 (1), 247-53 PMID: 21613557

Wednesday, 1 June 2011

Fiber Up! Dietary Fiber and Mortality Risk

We hear quite a bit in the lay press about the potential benefits of dietary fiber including eating multiple daily servings whole grains,  fruits and vegetables.  I tend to be skeptical of many of these recommendations.  But after following some of the research related to the Mediterranean Diet,  I at least have my antenna (and my Google Reader) up on this issue.  A previous post on basics of the Mediterranean diet is here and a review of a study linking the diet to slower cognitive decline in older adults is here.

My Google Reader recently retrieved an online first article from the Archives of Internal Medicine by Dr. Park and colleagues from the National Cancer Institute entitled "Dietary Fiber Intake and Mortality in the NIH-AARP Diet and Health Study". 

This is a longitudinal correlational study of the association between self-reported dietary fiber intake and cause of death from the National Death Index Plus registry.  I wasn't aware of this study that includes cooperation with the American Association of Retired Persons or AARP.  AARP collaborated with the National Institute of Health on a survey completed by 567, 169 AARP members in 1995 and 1996.

The survey collected detailed personal dietary habit information and then scanned the National Death Index for deaths in those who had completed the survey.   The daily dietary fiber intake was calculated for each participant and split into quintiles (five groups each comprised of 20% of the sample) with those with the highest fiber intake (top 20%) compared to those with the lowest fiber intake (lowest 20%).  Rates of death for these two groups were examined.  Since people who tend to eat more nutritious diets tend to also have other healthy lifestyle behaviors, a variety of potential confounding variables were controlled, i.e. smoking status, age, race, education, marital status, baseline health status, alcohol consumption, etc.

During the follow up period, here is the estimated reduced death rates associated with being in the highest fiber intake groups (compared to lowest) with women in bold first followed (men):
  • Death from cardiovascular disease: 34% lower (24% lower)
  • Death from cancer: 4% (not statistically significant) lower, (17% lower)
  • Death from respiratory disease: 46% lower, (41% lower)
  • Death from infectious disease: 69% lower, (56% lower)
Risk of death from accidents did not differ by dietary fiber ranking.  However, the study numbers are pretty impressive.  Although there is pretty good data supporting dietary fiber and cardiovascular disease risk, there is limited study in other causes of death.  This study supports association between dietary fiber and lower cancer deaths in men, but not women.  The authors point out that many of the types of male-predominant cancers (i.e. head and neck, esophageal, liver, bladder and kidney) may be those where dietary fiber plays a role.  Another potential interpretation is that breast cancer is not influenced by dietary fiber.

I was surprised by the finding of reduced death in the respiratory and infectious disease categories.  There were many fewer deaths in these categories so the results need to be interpreted cautiously.  However, there is at least one study (Iowas Women's Health Study) showing dietary fiber reduced death from inflammatory disorders.  The authors point out something I didn't know, that high dietary fiber is associated with reduced levels of endogenous (the body's own) inflammation markers including lower C-reactive protein, interleukin 6 and tumor necrosis factor alpha receptor.  Inflammation may play a role in deaths due to asthma and other lung inflammatory conditions.

This study provides some additional intriguing data.  The authors broke down the source of dietary fiber into whole grains, vegetables, fruits and bean sources.  What they found was that the dietary fiber from grains (but not fiber from vegetables, fruits or beans) contributed the largest effect of the association of high fiber intake with reduced mortality.  And this is where the figure above comes in.  The figure shows that in a whole wheat kernel, the wheat bran and wheat germ pack the most fiber, minerals and chemicals called phytochemicals.  The authors propose that high dietary grain fiber is likely to include high intake of whole grain components such as wheat bran and wheat germ.

Again, these findings are only correlational.  They do not prove a cause and effect.  There may be some other factor contributing to this reduced mortality in the high fiber group.  This study does not prove putting people with a low fiber diet on a high fiber diet will reduce their risk of death during a period of follow up.  But my skepticism is fading so please excuse me now as I'm heading to the store for some bran cereal and a box of rolled oats.

Disclosure: I had not reached the minimum age for AARP membership in 1995 and did not participate in this survey.  I am now an AARP member primarily for motel/hotel discounts for AARP members.  I received no money from AARP, the American Bean Council or other source for writing this post. 

The figure in this post comes from Wikipedia Commons file authored by jkwchui.

Park, Y., Subar, A., Hollenbeck, A., & Schatzkin, A. (2011). Dietary Fiber Intake and Mortality in the NIH-AARP Diet and Health Study Archives of Internal Medicine DOI: 10.1001/archinternmed.2011.18

Tuesday, 1 February 2011

What is Grass Based Health?

Animals make us human.
Temple Grandin

. . . the Andamanese believe it is the possession of fire that makes human beings what they are and distinguishes them from animals.
Radcliffe-Brown, A. 1922.
The Andaman Islanders: A Study in Social Anthropology.
Cambridge, UK: Cambridge University Press.1

So, what does barbecue make us?

With his new opposing thumb and his king-sized cranium,
Man sallied forth with grace and savoir faire.
With Promethian desire he soon discovered fire,
And arson but a single step from there.
The wheel and gasoline, to the full-sized limousine,
Music, art and law are but a few,
But name what can compare to the artistry so rare of
the sparerib that has met the barbecue.
from The Big Band Theory. Mark Graham.2

I recently had the opportunity to attend the 7th Annual Cattlemen’s Workshop in La Grande, Oregon. It was a wonderful opportunity to connect with the region’s beef industry and learn from the ranchers, industry and University folks who were there. One lesson I learned is that “Grass Based Health” means something to everyone, but it may not mean the same thing to everyone. So I thought it was time to define what I mean by the phrase.


Grass Based Health is the concept that pasture-based livestock production systems are better for the animals, better for the farmers and ranchers, better for the land, and better for the communities they are a part of than the alternative livestock production systems. I’ll be covering all of these topics in future posts. But the fundamental principle of Grass Based Health is that our diet ought to be grass based, not grain based. Understanding and accepting this premise then forces the consideration, and re-consideration, of many other topics as well.

It’s obvious (or it ought to be!) that we lack the herbivores’ specialized ingestive and digestive anatomy that permits them to utilize cellulose. We depend on various animals to convert cellulose, the most common organic compound on Earth[3], into animal fat and high quality, complete protein. The majority of feed units consumed by all domestic livestock (beef cattle, dairy cattle, hogs and poultry, sheep and goats, horses and mules, and “other”) in the US in 1970 came from forage (54.4% of all feed units, 36.1 and 18.3 percent from pastured and harvested forage, respectively).[4] I’ll simplistically refer to all forage as “grass” from now on. A grain-finished steer spends most of his life eating grass, and is only fed grain as a portion of its diet during the finishing phase. Even then, its ration still contains grass. So, even today meat and dairy products can be described as “grass based.” I do believe that there is vast room for improving the production and utilization of grass in pasture-based production systems, and I’ll address that in future posts. 

Terms like “grass-fed,” “free-range,” “pastured,” and “organic” are all used to market food today. Quantitative and qualitative differences have been determined between grass-finished and grain-finished meats. Similar differences have been determined between the milk from dairy cows grazing pasture compared to those housed in confinement systems. Just how important are these differences? Future posts will deal with this subject, too. I am concerned, however, that the claims some make for these products are over-stated. Without addressing the biggest insult to human health – an oversupply of readily digestible refined carbohydrates – is the consumer likely to realize any of these benefits? (see What's the Limiting Factor?)

It is beyond dispute that the “natural” diet of mankind is one that is much higher in animal products and much lower in carbohydrate than what our current “experts” advise us to eat. The archeological evidence provided in the fossil record and the testimony of anthropologists concerning various hunter-gatherer cultures provides significant evidence regarding our ancestor’s diet. The ancients who 19,000 years ago produced the awe-inspiring Lascaux cave paintings in present-day southwest France were paying homage to what gave them sustenance – the auroch, the ancestor of our modern European cattle.

Loren Cordain (2000) published an analysis of the diets of hunter-gatherer populations whose diets had been assessed by anthropologists.[5] One in every five of these 229 populations subsisted on almost entirely hunting or fishing. More than 85 percent of their calories came from meat or fish, with some groups thriving entirely on meat and fish. Only 14 percent of these groups got more than half their calories from plant foods. Not a single one of these populations was exclusively vegetarian. 

Today we understand the need to avoid obesity and the chronic diseases that are associated with being overweight. The “experts” tell us that we can accomplish these goals by eating less and exercising more, and by eating diets that are low in fat and high in carbohydrates. Animal products must be restricted, according to this advice. In response to the expert’s recommendations, as published in the USDA Food Guide Pyramid, the American caloric intake averages 15% from protein, 33% from fat, and the balance from carbohydrates. In contrast, Cordain’s hunter-gatherers’ diets were high to very high in protein (19 to 35 percent of calories) and high to very high in fat (28 to 58 percent of calories). Several of these populations obtained as much as 80 percent of their calories from fat. 

There is an abundance of evidence documenting the oft-repeated experience of isolated populations who exhibited none of the various “western diseases” until the introduction of sugar and white flour to their diets (see Grass and Cancer, Good Calories, Bad Calories, and Why we Get Fat and What to Do About It, among other sources). The scientific research and observational data so strongly supports the fattening carbohydrate hypothesis and explains the phenomenon of metabolic syndrome that all but those with a vested interest ought to be convinced that the lipid hypothesis should never have been adopted as the basis of public health policy in this country, or anywhere else. And this flawed “conventional wisdom” about weight loss and what constitutes a healthy diet has contaminated all of science. Not just nutrition and human health, but disciplines as seemingly unrelated as soil conservation. I’ll write about that in the future, too.


At the end of the introduction to his newest book, Gary Taubes writes:[6]

In the more than six decades since the end of the Second World War, when this question of what causes us to fatten – calories or carbohydrates -  has been argued, it has often seemed like a religious issue rather than a scientific one. So many different belief systems enter into the question of what constitutes a healthy diet that the scientific question – why do we get fat” – has gotten lost along the way. It’s been overshadowed by ethical, moral, and sociological considerations that are valid in themselves and certainly worth discussing but have nothing to do with the science itself and arguably no place in a scientific inquiry.

Carbohydrate-restricted diets typically (if not, perhaps, ideally) replace the carbohydrates in the diet with large or at least larger portions of animal products – beginning with eggs for breakfast and moving to meat, fish, or fowl for lunch and dinner. The implications of that are proper to debate. Isn’t our dependence on animal products already bad for the environment, and won’t it just get worse? Isn’t livestock production a major contributor to global warming, water shortages, and pollution? When thinking about a healthy diet, shouldn’t we think about what’s good for the planet as well as what’s good for us? Do we have a right to kill animals for our food or put them to work for us in producing it? Isn’t the only morally and ethically defensible lifestyle a vegetarian one or even a vegan one?

These are all important questions that need to be addressed, as individuals and as a society. But they have no place in the scientific and medical discussion of why we get fat.

It is understandable that Taubes would limit his argument, especially in this book which was envisioned as a simplified and focused discussion of the causes and treatment of obesity. Taubes is to be commended for acknowledging the broader ramifications of this subject and stating that, while he’s aware of these issues he would not address them. But there’s an implication in his statement that I’d like to dispute. 

I believe that we can address, scientifically, the questions regarding the impacts of animal agriculture on “the environment,” including the issues of anthropogenic global climate change (“global warming”), water quality and water quantity. The effort will show that this science is as muddled as Taubes found the disciplines of diet, nutrition and human health to be. Some of this muddle is the result of dietary dogma contaminating the discussion. If you believe that eating animal products is bad for you, it’s easy to imagine that belief impacting your perception of environmental issues. If your environmental beliefs are informed by the incorrect 18th and 19th century image of pre-Columbian North America as an “unspoiled wilderness,” then activities by man to transform the environment are necessarily seen as degradation. If you’ve never thought of the impacts of field-crop agriculture and horticulture, it might be possible to remain ignorant of the fact that greater soil erosion occurs in annual cropping systems that in perennial pasture-based animal production systems. Pasture-based livestock production systems protect surface and ground water quality. Credible science supports the role of well-managed animal agriculture in not only protecting but actually improving the environment. Future posts will discuss these issues. This, too, is what I mean by Grass Based Health.

In the Introduction to his book, Meat: A Benign Extravagance, Simon Fairlie makes the following statement:[7]

I am not overly concerned with questions of dietary health, nor do I take any interest in the diet and dentition of our remote ancestors.

Only someone not convinced of the fundamental requirement for animal fat and protein in the human diet could label meat an “extravagance!” Fairlie’s book is one of many I’m working my way through. His book apparently is having an impact. Folks who’ve been committed to vegetarianism are being swayed by his argument that animal products can be produced in ways that are sustainable. Glad to hear it. I remember being told almost twenty years ago that animal agriculture had no place in “sustainable agriculture.” “Organic” and “sustainable” were once synonymous with vegetarian.

My initial arguments with Fairlie’s thesis are that:
  • The vast majority of the earth’s surface is best suited for producing grass for grazing.
  • A significant portion of the “grain” that is fed to livestock are by-products like brewer’s grains and oil meal. These are not suitable for humans, and not utilizing them as livestock feeds would increase the cost of the primary products.
  • The field and horticulture crops are the “extravagance,” not the animal products. And they are far from “benign.” Soil loss through erosion and soil organic matter depletion, and water quality degradation, fossil fuel use, petrochemical inputs, wildlife impact – all of these are greater in annual cropping situations than in perennial pasture-based livestock production systems. And that list does not include the impact these crops have on the human health and the cost of treating the resulting diseases.
Consider diabetes: Diabetes is one of the western diseases, now understood to be part of metabolic syndrome. The US population today is approximately 312 million people, 76.9% of whom are adults (approximately 240 million). According to the Center for Disease Control and Prevention, 105 million adults in the US have diabetes or prediabetes (26 million with diabetes, 79 million with prediabetes), as diagnosed with hemoglobin A1c.[8] Thus diabetic and pre-diabetic adults represent 44% of the US adult population. The increase in those with pre-diabetes, now equal to 1 in 3 US adults, represents a 39 % increase since 2008. The recommended plant-based diet can hardly be called benign! The CDC estimates the direct and indirect costs of diabetes at $179 billion annually. The recommended plant-based diet can hardly be called sustainable, either.

I sometimes hear those in the low-carb / Primal / Paleo community refer to “agriculture” negatively. It is important to remember that the farmers and ranchers – the people who actually steward natural resources to produce our food - have been taken in by the same official dietary guidelines as the rest of us. They are challenged by the same chronic health problems the rest of us face. They need to hear this alternative to the conventional wisdom. The average age of farmers in the US is just over 57 years.[9] Sustainability implies longevity, but the intergenerational transition of the current production models have been difficult to achieve. Is there an alternative? There can be no sustainability without profit. In western Oregon, traditional dairies are finding it difficult to break even while pasture-based dairies are making a profit. Dairymen from Europe and New Zealand have been looking at the potential for grass-based dairying in the US. Some have done more than just look. Americans spend less, as a percent of their income, on food than any other industrial country. And we spend more on medical care. We can pay our farmers or we can pay our doctors. Payin’ our doctors hasn’t worked out all that well. That, too, is what Grass Based Health is about.

Finally, if meaningful change is going to happen in our nutrition policies and all that they influence it will have to begin at the grass roots. Individuals will have to learn on their own what we should have been taught since the 1960s. They will have to apply this information to their own lives and obtain the thoroughly predictable results – improved health and weight loss. They will then pass the word along to their families and friends, some of whom will listen and adopt this appropriate way of eating and living. And so it will spread until we reach the tipping point. A grass roots movement. But "Grass Roots Health” was already taken (and a worthwhile effort that is, too, regarding the role of vitamin D in preventing chronic diseases!). 

So now you know I called this blog “Grass Based Health,” what I mean by that phrase, and some of what I’ll be writing about in future posts.



[1] From Wrangham, R. 2009. Catching Fire: How Cooking Made Us Human. New York, NY
[2] Mark Graham. 1979 in The Mark Graham Songbook: Twenty-Five Originals From the Forked-Tongue Demon. Mark Graham. 1991.
[3] Cellulose. (2008). In Encyclopædia Britannica. Retrieved January 11, 2008, from Encyclopædia Britannica Online.
[4] Meath, M. E., D. S. Metcalfe, R. F. Barnes. 1973. Forages; The Science of Grassland Agriculture. The Iowa State University Press. Ames, Iowa.
[5] Cordain, L., J. B. Miller, S. B. Eaton, N. Mann, S. H. Holt, and J. D. Speth. 2000. “Plant-Animal Subsistence Ratios and Macronutrient Energy Estimations in Worldwide Hunter-Gatherer Diets.” American Journal of Clinical Nutrition. Mar;71(3):682-92.
[6] Taubes, G. 2011. “Why We Get Fat and What To Do About It.” 2011. Alfred A. Knopf, New York. pp 11-12
[7] Fairlie, S. 2010. “Meat: A Benign Extravagance” Chelsea Green Publishing Co.
[8] Centers for Disease Control and Prevention. 2011. National Diabetes Fact Sheet: National Estimates and General Information on Diabetes and Prediabetes in the United States 2011. Atlanta, Ga.: U.S. Department of Health and Human Services, Centers for Disease Control and Prevention.
[9] http://www.agcensus.usda.gov/Publications/2007/Online_Highlights/Ag_Atlas_Maps/Operators/Characterists/07-M124-RGBChor-largetext.pdf